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He noticed something remarkable during his research: when aged liver tissue was exposed to GHK-Cu, it began synthesizing proteins in patterns that resembled much younger tissue

However, it was not clear how ASA exerts its multiple effects in oxidative stress conditions when cellular GSH pool is altered (depleted or enhanced)

The answer is short and simple: We dont know

Multiple cellular processes are considered to contribute to this functional decline and these are collectively referred to as the hallmarks of aging and these include processes like genomic instability, telomere attrition, epigenetic alterations, loss of proteostasis, disabled macroautophagy, deregulated nutrient-sensing, mitochondrial dysfunction, cellular senescence, stem cell exhaustion, altered intercellular communication, chronic inflammation, and dysbiosis 1

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